Coming into the 2015 season, the battle for the starting goalkeeper position for the University of Wisconsin-Madison men’s soccer team looked like it was going to be a two-man race.Redshirt junior Casey Beyers and sophomore Adrian Remeniuk split game time last season, playing in nine and 11 games respectively. Beyers started the season as the No. 1 option, but Remeniuk also saw game time in the second half of the Badgers 2-0 loss to Xavier on Aug. 30.But a new goalkeeper has emerged from the ranks to contend for the starting role along with Beyers and Remeniuk: redshirt junior Connor Revsbeck.Revsbeck began the season as third string, but the Lakeville, Minnesota native started his first two games in his Wisconsin career on Sept. 4 and Sept. 6 as the Badgers lost both matches to Santa Clara, 2-1 (OT), and UC-Davis, 3-2.Revsbeck was happy to finally get his chance between the sticks for the Badgers, even if it was in a losing fashion.“It was nice,” he said. “Obviously the results didn’t go our way, but it was good to go out there and help support the team.The shot stopper didn’t even know he was going to be starting two weekends ago. Revsbeck said he found out he was getting the nod for both games when the Badgers landed in California, giving him roughly a day to prepare for his first career start for Wisconsin.Revsbeck transferred from UW-Milwaukee after his freshman season. He made five appearances for the Panthers, and couldn’t compete for more playing time due to injuries. He said he’s trained hard to get healthy and give himself the opportunity to play.Revsbeck said he transfered from UW-Milwaukee not because he didn’t like playing for the team there, but rather for a greater challenge academically, Revsbeck said.Head coach John Trask was pleased with the way Revsbeck performed in his first two appearances for the Badgers.“I thought [Revsbeck] had a gutsy performance, especially [against Santa Clara],” Trask said. “He made a couple of excellent saves to keep us in the game.”Compared to Beyers and Remeniuk, Trask said Revsbeck is likely the quickest of the goalkeepers, due in part to his shorter stature — standing at 6-foot-2, while Byers towers at 6-foot-5.“[Revsbeck]’s quick — more of a reaction save type of guy,” Trask said. “We thought that in the situation out West, we would need a little bit more than a dominant force in the box.”Beyers and Remeniuk are top goalkeepers in their own respect, each bringing something different to the table. Beyers offers the dominant, looming force that Trask talked about. Beyers rarely loses aerial battles in the box, often using his large frame to snag balls out of the air before the on-rushing heads of attackers can get a piece of it.Remeniuk, one of most talented sophomore goalkeepers in the college game, uses his athleticism and communication skills to make his presence known when he’s busy patrolling the box for the Badgers.Last season for Wisconsin, Beyers posted a 1-5-2 record, had a 2.0 goals against average, racked up 33 saves and notched one shutout in his eight appearances. Remeniuk, on the other hand, managed a 2-7-1 record, had a 1.6 goals against average, recorded 33 saves and posted two shut outs in his 10 appearances.So far in the 2015 season, each of the three goalkeepers have seen minutes, but no one has stood out thus far to solidify that No. 1 role. This has left Trask with the predicament of deciding which keeper is playing the best at the moment.“As they say in hockey, you’re going with the hot goalkeeper,” he said.The Badgers have already conceded eight goals in four games this season, something that needs to be addressed for Wisconsin. In order for that to happen, however, it all starts with the goalkeeper, said Revsbeck.“We need to communicate more at the back line and make sure everybody’s one cohesive unit and just go from there,” he said. “Hopefully we’ll start getting more results.”Beyers and Remeniuk are likely to get most of the minutes the rest of the season, especially with the start of Big Ten play on the horizon, Trask said, but Revsbeck made the most of his opportunity and will look to continue to push the level of play of the other goalkeepers.
It’s no secret that a mouse stops growing before it becomes the size of a whale. But the physiological and genetic mechanisms that control how fast—and how big—an animal grows have largely remained a mystery. A study of fruit flies, however, is shedding some new light on how animals regulate body size, and may also help explain why certain cancer tumors grow out of control.Two largely independent processes determine how big an animal gets. The first controls how fast an animal grows and depends on insulin or similar hormones to cue cells to grow and divide. The second regulates the timing of maturation or, in insects, metamorphosis (the process by which a caterpillar turns into a butterfly). In fruit flies, for instance, two hormones influence the timing of metamorphosis. One is ecdysone, known as the molting hormone because elevated levels can prompt an insect to shed an old skin or undergo other changes. The other is juvenile hormone (JH), which stops growth and initiates metamorphosis when levels drop.Past studies have shown that researchers can influence insect size by tinkering with these hormonal pathways. In a 2010 study, for instance, scientists created fruit flies unable to produce growth-stopping JH. The result: stunted adults that were only 75% of the usual size, many of which died before reaching adulthood. But it wasn’t clear why. One possibility was that the loss of JH shortened a key growth phase and triggered premature metamorphosis, causing smaller larvae to transform into smaller adults. Alternatively, the loss of JH might have simply slowed the rate of growth but not changed the timing of metamorphosis.Sign up for our daily newsletterGet more great content like this delivered right to you!Country *AfghanistanAland IslandsAlbaniaAlgeriaAndorraAngolaAnguillaAntarcticaAntigua and BarbudaArgentinaArmeniaArubaAustraliaAustriaAzerbaijanBahamasBahrainBangladeshBarbadosBelarusBelgiumBelizeBeninBermudaBhutanBolivia, Plurinational State ofBonaire, Sint Eustatius and SabaBosnia and HerzegovinaBotswanaBouvet IslandBrazilBritish Indian Ocean TerritoryBrunei DarussalamBulgariaBurkina FasoBurundiCambodiaCameroonCanadaCape VerdeCayman IslandsCentral African RepublicChadChileChinaChristmas IslandCocos (Keeling) IslandsColombiaComorosCongoCongo, The Democratic Republic of theCook IslandsCosta RicaCote D’IvoireCroatiaCubaCuraçaoCyprusCzech RepublicDenmarkDjiboutiDominicaDominican RepublicEcuadorEgyptEl SalvadorEquatorial GuineaEritreaEstoniaEthiopiaFalkland Islands (Malvinas)Faroe IslandsFijiFinlandFranceFrench GuianaFrench PolynesiaFrench Southern TerritoriesGabonGambiaGeorgiaGermanyGhanaGibraltarGreeceGreenlandGrenadaGuadeloupeGuatemalaGuernseyGuineaGuinea-BissauGuyanaHaitiHeard Island and Mcdonald IslandsHoly See (Vatican City State)HondurasHong KongHungaryIcelandIndiaIndonesiaIran, Islamic Republic ofIraqIrelandIsle of ManIsraelItalyJamaicaJapanJerseyJordanKazakhstanKenyaKiribatiKorea, Democratic People’s Republic ofKorea, Republic ofKuwaitKyrgyzstanLao People’s Democratic RepublicLatviaLebanonLesothoLiberiaLibyan Arab JamahiriyaLiechtensteinLithuaniaLuxembourgMacaoMacedonia, The Former Yugoslav Republic ofMadagascarMalawiMalaysiaMaldivesMaliMaltaMartiniqueMauritaniaMauritiusMayotteMexicoMoldova, Republic ofMonacoMongoliaMontenegroMontserratMoroccoMozambiqueMyanmarNamibiaNauruNepalNetherlandsNew CaledoniaNew ZealandNicaraguaNigerNigeriaNiueNorfolk IslandNorwayOmanPakistanPalestinianPanamaPapua New GuineaParaguayPeruPhilippinesPitcairnPolandPortugalQatarReunionRomaniaRussian FederationRWANDASaint Barthélemy Saint Helena, Ascension and Tristan da CunhaSaint Kitts and NevisSaint LuciaSaint Martin (French part)Saint Pierre and MiquelonSaint Vincent and the GrenadinesSamoaSan MarinoSao Tome and PrincipeSaudi ArabiaSenegalSerbiaSeychellesSierra LeoneSingaporeSint Maarten (Dutch part)SlovakiaSloveniaSolomon IslandsSomaliaSouth AfricaSouth Georgia and the South Sandwich IslandsSouth SudanSpainSri LankaSudanSurinameSvalbard and Jan MayenSwazilandSwedenSwitzerlandSyrian Arab RepublicTaiwanTajikistanTanzania, United Republic ofThailandTimor-LesteTogoTokelauTongaTrinidad and TobagoTunisiaTurkeyTurkmenistanTurks and Caicos IslandsTuvaluUgandaUkraineUnited Arab EmiratesUnited KingdomUnited StatesUruguayUzbekistanVanuatuVenezuela, Bolivarian Republic ofVietnamVirgin Islands, BritishWallis and FutunaWestern SaharaYemenZambiaZimbabweI also wish to receive emails from AAAS/Science and Science advertisers, including information on products, services and special offers which may include but are not limited to news, careers information & upcoming events.Required fields are included by an asterisk(*)To sort it out, a team led by developmental biologist Christen Mirth of the Instituto Gulbenkian de Ciência in Oeiras, Portugal, and fly geneticist Alexander Shingleton of Lake Forest Collegein Illinois bred both normal and JH-deficient flies, and then compared a suite of characteristics, including the timing of growth and levels of growth hormones. JH-deficient flies did not metamorphose prematurely, suggesting that the second hypothesis was correct, the team reports online this week in the Proceedings of the National Academy of Sciences. Surprisingly, however, the researchers also found that they could eliminate the stunting effect by altering a JH-deficient fly’s ability to produce the other two growth-related hormones. A JH-deficient fly would grow to normal size, for example, if they used genetic techniques to boost ecdysone levels or prevent it from turning off insulin production.This extensive crosstalk between the hormones was surprising, Mirth says. It appears that “JH acts to fine-tune growth rates” by influencing the effects of the other two hormones, she says.If the findings hold true in other kinds of insects and animals, they could help reveal universal mechanisms of growth and size regulation, says Yuichiro Suzuki, an assistant biology professor at Wellesley College in Massachusetts, who was not involved in the work.The hormonal crosstalk may also help researchers better understand cancer tumors in humans. That’s because similar hormones appear to be involved in triggering the runaway cell replication that causes certain kinds of cancers, such as breast, ovarian, prostate, and testicular cancer. By learning to tinker with the hormonal pathways, the authors suggest, doctors might one day be able to prevent mouse-sized tumors from reaching elephantine proportions.